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Future Perspectives The scientific studies on seleno-GPxs have so far led to the understanding that the physiological role of seleno-GPxs is not only limited to detoxification of peroxides, but also involves regulating signaling pathways (such as chromatin remodulating, NF-B activation, insulin signaling, mitochondrial integrity, and ferroptosis, among others) [1, 8,9,10]

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VSMC death causes vascular inflammation, loss of extracellular matrix (ECM) and collagen, weakening of the fibrous cap, and ultimately plaque rupture (Grootaert and Bennett, 2021

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