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It is a coenzyme of Niacin (Vitamin B3), which acts as a helper molecule that binds to a protein in order to activate an Enzyme reaction

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Therefore, the presence of immune exhaustion in infectious syndromes could be a compensatory mechanism to slow-down the progression of chronic infection, as occurs with AIDS patients (234, 236, 238)

This result suggests that SMN expression in blood may not be a good surrogate biomarker to track VPA response

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& Dallner, G