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inhibition of GPX4 induces ferroptotic cell death in these cells
Becerril S, Frhbeck G
The canonical activation pathway is triggered through the site-specific phosphorylation by an IB-kinase (IKK) complex in response to microbes, and proinflammatory cytokines
According to this study (137), it has been shown that in UC, the activation of the iNOS/COX-2/5-LOX loop and increased levels of their end products, such as NO, prostaglandin E2 (PGE2), and leukotriene B 4 (LTB 4), which lead to the overproduction of free radicals and the impairment of the antioxidative system
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