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The interplay between -synuclein pathology, neuroinflammation, dysfunction of mitochondria, oxidative stress, and altered protein degradation pathways culminates in neuronal degeneration

Coming back to the mentioned general theoretic cytoprotection effects (Robert, 1979

10.1016/j.biomaterials.2012.12.009 [DOI] [PubMed] [Google Scholar] Charlier E., Deroyer C., Ciregia F., Malaise O., Neuville S., Plener Z., et al

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Roles of the peroxisome proliferator-activated receptors (PPARs) in the pathogenesis of nonalcoholic fatty liver disease (NAFLD)