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Every body composition claim about the compound extrapolates from preclinical mouse data

However, it remains to be determined whether its beneficial effects are primarily mediated via the inhibition of direct PPAR target genes, such as CD36, or through the PPAR-mediated repression of proinflammatory transcription factors, such as NF-kB

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doi: 10.1016/j.celrep.2018.10.080 68 KochB.TravenA

In the pre-treatment studies, NaBPC157 delayed and prevented the arrhythmia occurrence and reduced and/or prevented ischemia