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doi: 10.1007/s10741-018-9716-x 33

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10.1210/jc.2009-1579 3 BegleyE.McVeighJ.HopeV.BatesG.GlassR.CampbellJ.et al (2017)

In all patterns of ethanol consumption described above, we reported changes associated with mitochondrial injury [11, 27, 132], therefore strongly suggesting that mitochondria is an important mediator of ethanol neurotoxicity and could be considered as a potential therapeutic approaches for treating ethanol-associated disorders

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