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The study result indicated its most likely because of new protein synthesis and the activation of the mTOR pathway (a driver for increased muscle mass)

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The majority of these combinations induced mitochondrial dysfunction

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while the symptomatic acute COVID-19 condition could manifest into 3 clinical phases: (i) hypoxia and hypoxemia (Warburg effect), (ii) hyperferritinemia (cytokine storm), and (iii) thrombocytosis (coagulopathy)