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In heterozygotes, mutated chains interfere with the assembly of anchoring fibrils by normal type VII collagen chains

These cells, through complex interactions, guarantee the BBBs integrity and the normal metabolic state of neurons [2, 3]

Klicek et al., 2008

Psychiatry 130, 110915

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By elevating extracellular glycine levels, GlyT1 inhibition enhances NMDA receptormediated neurotransmission, potentially addressing cognitive deficits and synaptic dysfunction commonly observed in Alzheimers and related dementias