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This action preserves membrane integrity and prevents the lethal cascade that ferroptosis triggers

VRK2 overexpression could suppress the increase of shrunken mitochondria and lipid ROS induced by TBHP, while BFA treatment abolished VRK2-mediated alterations (Fig

Reusch, J

In this context, oxidative stress plays a significant role by increasing ROS levels in renal cells, which damages lipids, proteins, and DNA

Peroxisome proliferator activator receptor (PPAR)- Ligand, but Not PPAR- , ameliorates cyclophosphamide-induced oxidative stress and inflammation in rat liver