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Red blood cells homeostatically bind mitochondrial DNA through TLR9 to maintain quiescence and to prevent lung injury

Matrix and repair signalling layer GHK Cu is mapped to extracellular matrix readouts

This releases nuclear factor-B (NF-B) heterodimers (typically p50/p65) from cytoplasmic sequestration, allowing their nuclear translocation where they bind to B response elements in target gene promoters

By re-expressing GSTP1, increased detoxification, and reduced levels of oxidative stress could potentially contribute to reduced prostate cancer progression and even to an abrogation on evolution toward invasive and metastatic disease

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