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A final consideration is that second messenger-dependent protein kinases could phosphorylate other proteins, which subsequently influence GPCR desensitization, such as regulator of G protein signalling proteins (Cunningham et al ., 2001) or the Raf kinase inhibitor protein, which upon PKC phosphorylation becomes able to inhibit GRK2 function and hence -AR desensitization (Lorenz et al ., 2003)

PLoS Med 14, e1002261.Google Scholar Foster, TC, DeFazio, RA, Bizon, JL

Scientific Reports, 5 (1), 1-9.

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doi: 10.1073/pnas.0701549104 54 ZenattiPPRibeiroDLiWZuurbierLSilvaMCPaganinMet al