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This complex biological process that causes clinical symptoms, including heat, pain, redness, and swelling, is broadly triggered by infection and tissue damage [2]
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In parallel, electrophilic oleic acid derivatives such as nitro-oleic acid (oleic acid-NO) have shown renoprotective actions in tubular and glomerular injury models via Nrf2-driven antioxidant programs and suppression of NADPH oxidase activity, illustrating that oleic acid-derived lipid mediators can engage cytoprotective signaling networks distinct from those activated by lipotoxic oleic acid overload (Nie et al
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