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Acting preventively, BPC-157 prevents congestion and venous insufficiency and will be a new precursor in supporting the treatment of thrombotic disease
Several limitations of this study warrant consideration
Investigators have proposed that GHK-Cu's central function is to deliver copper to fibroblasts in a controlled, non-toxic form buffering against the redox damage that free Cu would otherwise cause [4]
However, the endothelial damage observed in ME/CFS patients does not appear to correlate with the usual markers of endothelial dysfunction seen in cardiovascular disease such as increased levels of symmetric dimethylarginine (SDMA), asymmetric dimethylarginine (ADMA), and high-sensitivity C-reactive protein (hs-CRP), and reduced levels of arginine and homoarginine (311)
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