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4 Impaired excretion is most often due to abnormalities in the kidney urate transporter (called URAT1) or organic ion transporter (OAT), both of which control the movement of uric acid out of proximal kidney tubules and into urine

It uses ATP from mitochondria for this, and blocking P-gp with diltiazem brings back gemcitabines toxicity (Gu et al., 2022)

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Table 2), and this was improved following treatment with Fer-1 (26.81 2.71 g

GSH biosynthesis was identified by the mRNA levels of GshF , a critical amino acid ligase of the bifunctional pathway