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Abstract Background: The widespread clinical use of immune checkpoint inhibitors (ICIs) in oncology has been accompanied by an increased incidence of immune-related adverse events (irAEs)

Ayala A, Munoz MF, Arguelles S

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The sensitivity of the DNA damage checkpoint prevents oocyte maturation in endometriosis

The increased oxidative stress seems to be the second hit in the progression from NAFL to NASH: the increase in ROS and consequently in lipid peroxidation has been associated to hepatocellular damage, inflammation, and eventual fibrosis due to the activation of hepatic stellate cells