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doi: 10.1016/j.tice.2025.102837

Similarly, pS6K levels were significantly upregulated in HG-treated HRMC cells and improved by HU treatment (Figures 7C, D)

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Moreover, the findings that mice with neutrophil-specific Gpx4 haploinsufficiency recapitulate key clinical features of human SLE, including autoantibodies, neutropenia, skin lesions and proteinuria, and that the treatment with a specific ferroptosis inhibitor significantly ameliorates disease severity in lupus-prone mice reveal the role of neutrophil ferroptosis in lupus pathogenesis

Featherstone DE, Shippy SA

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