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Thus, l-proline is an inhibitor of the 5OP aggregation response likely because of the structural resemblance between the two molecules

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Therapeutics that block RSL3-mediated GPX4 inactivation may provide a targeted approach to preserving antioxidant capacity in ARDS patients, reducing the extent of ferroptosis-driven lung damage ( 5.3 Iron chelators Iron overload exacerbates ferroptosis by fueling ROS production and lipid peroxidation, making iron chelation a viable therapeutic approach

10.22201/facmed.20075057e.2020.35.20205 Investig