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Like UQCRC1 in preventing cytochrome c from release, functions of ETC proteins beyond oxidative phosphorylation might also contribute to the pathogenesis of PD

Formulated for advanced studies examining metabolic regulation, mitochondrial activity, and peptide signaling pathways in model systems

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In advanced cellular assays, this has been shown to increase mitochondrial biogenesis and enhance the sirtuin-mediated stress response, as documented in prominent metabolic research databases

Cancers , 10 (8), 253