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Increasing secretion of pro-inflammatory chemokines and cytokines (e.g., IFN-, CXCL9, and CXCL10), together with upregulation of NKG2D ligands (e.g., MICA and ULBP), MHC class molecules, and decreasing local IP guardians lead to the breakdown of hair follicle IP [5]

Novel concepts of neuropeptide-based drug therapy: vasoactive intestinal polypeptide and its receptors

Histologic features of hepatitis B are inflammation that is usually around the portal tract, variable fibrosis that initially is also portocentric, and the presence of ground-glass hepatocytes

Neural mechanisms, such as neurogenic inflammation, are also important in NHIC, where nerve activation exacerbates bladder pain

This is well documented

H., MacKintosh, C