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Cagrilintide is an amylin analogue, not a GLP-1 receptor agonist, so it works through a distinct hormonal pathway

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Nov 20 2021;17(1):358

Established treatments (e.g., metformin, thiazolidinediones, lifestyle interventions) already modulate metabolic and inflammatory axes and may have indirect effects on neutrophil priming and NETosis

Increasing evidence supports the involvement of DNA methylation in RA development, bridging the gap between genetic variations and patient outcomes [31]

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