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Recent studies show EVs inhibit ferroptosis through multiple pathways to improve diabetic wound healing

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It was shown recently that treatment of erythrocytes with bacterial neuraminidase leads to desialylation of the glycocalyx and provokes cell surface PS exposure [216]

In addition to the tumorigenic effects of succinate accumulation, SDH5 mutation is the key driver supporting the acquisition of epithelialmesenchymal transition (EMT) characteristics

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Anergic T cells are metabolically anergic