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These pathways can provide important compensation when GPX4 is partially impaired, also indicating that ferroptosis regulation has high redundancy, and single-target intervention may not completely block the pathological process

& Sternberg, P

These actions are relevant in research studying emotional regulation, anxiety response, and central neurotransmitter turnover

In Alzheimers disease, the production of pro-inflammatory factors and A can over-activate microglia, secrete inflammatory factors and neurotoxins, and then induce neuronal damage and even apoptosis, thereby triggering Alzheimers disease