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Magalhes, L., Segundo, M., Reis, S., Lima, J., Tth, I., & Rangel, A

[DOI] [PMC free article] [PubMed] [Google Scholar] 72.May M.J., Leaver C.J

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These infants required ventilatory support and high concentrations of supplemental oxygen therapies that induced lung damage with regions of atelectasis and regions of hyperinflation, epithelial injury, hyperplasia of airway smooth muscle, fibrosis, and pulmonary vascular hypertension

However, the aforementioned studies did not elucidate the relative propensities of GLRXs cysteines to react with H 2 O 2 or GSSG, nor did they reveal how oxidation of each cysteine, either alone or in combination, contributes to inactivation of GLRX