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An adequate and hopefully incisive therapeutic action will therefore depend on a finely orchestrated balance between the efficacy of the single treatment for the specific pathogenetic mechanism and the control of potentially noxious interactions among the different therapies

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The table also details novel mechanisms, including the roles of Alas2 overexpression [94], SPRY4-ERK1/2 phosphorylation [107], and the interplay between p66Shc-Sirt1 [54], the ADMA-DDAH1 axis [56], and Hypoxia-Inducible Factor 1-alpha (HIF-1) signaling [105]

A consultation is required to determine suitability for treatment