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This could suggest an overactive Gln-Glu cycle in adults with ASD, which may represent either a manifestation of, or a compensatory response to, dysregulated excitatory neurotransmission implicated in the pathophysiology of ASD

63 In brief, the different modulation of PGC-1 by Akt isoforms may be due to diverse regulatory levels and cellular processes, and more comprehensive investigation regarding the exact mechanism of Akt isoforms in regulating PGC-1 are required

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