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Graphical abstract Similar content being viewed by others Introduction Wilsons disease (WD), which leads to excessive accumulation of copper, is an autosomal recessive disorder with a potentially life-threatening outcome and an incidence rate of 1:30,000 [1]

The complex is eventually released from lysosomes and transported across the cell membrane bound to transcobalamin in the blood circulation

62 Using transgenic mice, we showed pressor and renal vasoconstrictor responses to Ang IV to be mediated by the AT 1a receptor subtype