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Oxidative stress, an imbalance between reactive oxygen species (ROS) and antioxidant defense mechanisms in the cell, is involved in several physiological and pathological processes, including carcinogenesis
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Our data suggested that mutation of 3UTR-1 had no obvious effect to rescue the phenotype caused by DDX3X or METTL16 deficiency, whereas mutation of 3UTR-2 or 3UTR-3 fully eliminated the outcome induced by DDX3X or METTL16 inhibition (Fig
