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Lung injury and lung cancer caused by cigarette smoke-induced oxidative stress: Molecular mechanisms and therapeutic opportunities involving the ceramide-generating machinery and epidermal growth factor receptor

meningitidis can inhibit the intrinsic apoptosis pathway, translocating its porin PorB to the voltage-dependent anion channel (VDAC) on the mitochondrial outer membrane to prevent cytochrome c release (Massari et al., 2000, Massari et al., 2003)

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