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PMID: 30140235

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Suitable treatment could have promising eradication rates without recurrence of infection

The above evidence suggests that either the use of exogenous antioxidants or activation of the in vivo antioxidant enzyme system to eliminate excess ROS in the TME and maintain the balance between ROS and antioxidants can inhibit the T cell exhaustion program, promote the differentiation of memory-like phenotypes, and restore proliferative and effector functions, thereby enhancing anti-tumor immune effects

Nat Protoc 6:743760 Yale SH, Mazza JJ (2000) Anion gap acidosis associated with acetaminophen