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The authors concluded that GHK-Cu accelerates wound healing and skin repair through coordinated effects on collagen and glycosaminoglycan turnover, modulation of metalloproteinase activity, attraction of immune and endothelial cells to injury sites, and restoration of replicative vitality in irradiated fibroblasts [1]

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In the APP (Swe)/[PS1(L166P)] transgenic (Tg) model of AD, germ-free status reduced cerebral A load compared to conventionally raised APP rodents (Harach et al., 2017), implicating microbiota as a direct or indirect modifier of peripheral amyloidosis (discussed later in Section Gut Microbiome-Mediated Fibril Formation)

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