It also induces the reverse transport of these transporters (Sulzer et al., 1995), leading to the release of monoamines from the neuronal cytoplasm into the synaptic cleft ( 2.1.2 Vesicular monoamine transporter 2 inhibition METH inhibits vesicular monoamine transporter 2 and disrupts monoamine storage in synaptic vesicles ( 2.1.3 Calcium dysregulation and excitatory neurotransmission METH enhances glutamate release, leading to NMDAR overactivation and increased calcium influx into neurons (Staszewski and Yamamoto, 2006)
However, NAC did not significantly impact other factors like the number of follicles, endometrial thickness, or progesterone levels
Notably, despite theoretical concerns about increased infection risk due to neutrophil protease inhibition, no signal for increased severe infections emerged
10.17116/jnevro20151155157-63 Zh Nevrol
Allocati, N., Masulli, M., Di Ilio, C
IJPR 12 (1), 123129