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In contrast, the GSH-treated samples appeared relatively normal, and their mitochondria were characterized by abundant, large, and clear cristae (Fig

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We observed that the presence of IGF-1 significantly increased CD107a release in human CD34 + -derived CD56 + NK cells compared with that of CD56 + effectors derived from the same CD34 + population in the presence of SCF, Flt3-L and IL-15 alone (Fig

Estrogens also signal through membrane-bound receptors (e.g., GPER1 and palmitoylated-ER variants) to initiate non-genomic signaling, and these pathways often intersect and cross-regulate each other (145148)

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