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Notably, when combined with immunosuppressive drugs, Clostridium s immunomodulatory effects may enhance immune activity and compromise drug efficacy, requiring close monitoring of immune function and timely dosage adjustments

Following the activation of pro-inflammatory M1 macrophages, the activation of NF-B, upregulation of HIF-1, and the presence of ROS synergistically contribute to the polarization of macrophages towards the M1 phenotype (74)

Treatment discontinuation due to side effects occurred in 4.3%, 7.1%, 6.2%, and 2.6% of participants in the 5, 10, 15 mg, and placebo groups, respectively [18]

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These enzymes are deemed essential for neutralizing harmful free radicals and maintaining cellular integrity