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Conclusion: Our results indicate a protective effect of IGF-1 against OA pathogenesis by inhibition of NF-B signaling via regulation of the MAPK and PI3K/Akt signaling pathways and prevention of apoptosis by suppression of ROS production

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Front Endocrinol 11:102 Qian Y, He L, Su A, Hu Y, Zhu J (2023) Generation of a mouse spontaneous autoimmune thyroiditis model

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