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One standout mechanism involves managing inflammation by guiding immune cells, specifically macrophages, from a pro-inflammatory state (M1 phenotype) to a pro-repair state (M2 phenotype)

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It decreases lipogenesis (fat creation) in the liver

(2022) used fluorescent probes containing lactyl-lysine residues and confirmed in glial cells that SIRT1, SIRT2, SIRT3, and SIRT5 can catalyze delactylation