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Overexpression of thioredoxin reductase 2 (TrxR2) in cancer cells is often considered a critical factor in the complex interplay of tumorigenesis, disease progression, and the resistance to apoptosis, underscoring its significant role in the intricate dynamics of cancer development and survival

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Mechanism of Action Modulates the nitric oxide (NO) system, upregulates growth factor expression (EGF, VEGF), and promotes angiogenesis and granulation tissue formation to accelerate wound healing and tissue repair

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doi: 10.1038/s41598-017-17310-4