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The c-MET receptor tyrosine kinase is overexpressed in H69 cells and is inducible by exogenous HGF, resulting in induction of tyrosine phosphorylation at the major autophosphorylation sites pY1230/1234/1235 in the catalytic kinase domain, and also the pY1003 site in the juxtamembrane domain of c-MET (Figure 1)

Hierarchy of Evidence: Level A (Human meta-analyses and systematic reviews of CNS drug delivery)

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