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Melatonin exerts protective effects in an ILC2-biased model of allergic airways disease, even in the presence of canonical receptor blockade To determine whether the protective effects of melatonin extended beyond allergen-driven disease, we next employed an ILC2-biased IL-33 model of allergic airways inflammation ( Figure 2A )

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Here, both Mtnr1a and Mtnr1b as well as Aanat and Asmt were again undetected, suggesting that ILC2s did indeed not express the genes for canonical melatonin receptors or those genes required for the synthesis of melatonin ( Supplementary Figure 3E )

for instance, it works as an antioxidant and anti-inflammatory agent, aids in the neutralization of free radicals, and assists in preventing injury to the cells due to oxidative stress and various related issues (3)

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J., Oldfield, S