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Understanding the neurochemical changes and molecular consequences of KP activation during insomnia and its contribution to other comorbid conditions is yet to be fully investigated

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The medicine used is Hydroxcobalamin 1000mcg/ml ampoule

Administration ofthe ROS inhibitor N- acetylcysteine (NAC) partially restored GSH, attenuated ROS accumulation, and modestly alleviated Gln-deprivation-induced growth inhibition (Fig