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In summary, accumulating evidence supports a critical role for ferroptosis in secondary injury after both TBI and SCI, driven by iron overload, lipid peroxidation, and dysregulated ferroptosis-associated proteins (114, 225235)

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We injected AAV2/Retro-hSyn-Cre virus into the small intestinal wall, as well as bilaterally infused AAV-DIO-hM3Dq-mCherry or AAV-DIO-mCherry control virus into the DMV of C57 mice (Fig

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