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10.1016/j.neuro.2007.01.014 18 DanemanR.PratA

The results showed that NAC treatment partially reversed the HT-induced decrease in cell viability, suggesting that HTs ability to inhibit the proliferation of HCT116 and SW480 cells may be related to the generation of its autoxidative effect

Moreover, administration of N-acetyl cysteine (NAC) or ascorbate, antioxidants that increase tissue glutathione levels, abolished ovariectomy-induced bone loss, while L-buthionine-(S,R)-sulphoximine (BSO), a specific inhibitor of glutathione synthesis, caused substantial bone loss

doi: 117

Cotinine halts the advance of Alzheimers disease-like pathology and associated depressive-like behavior in Tg6799 mice