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LOXs also activate immune cells through LOX-derived proinflammatory metabolites, including leukotriene B4 (LTB4), LTC4, LTD4, LTE4 and Hydroxyeicosatetraenoic acids (HETEs)[110], which indirectly lead to ferroptosis

In vitro promotion of cell proliferation, migration and angiogenesis effects of the G/D-CuP The formation of new blood vessels, called angiogenesis, is a crucial event during the proliferation stage of wound healing, and poor vascularization contributes to the delayed healing of diabetic wounds

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