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Upon binding of a ligand to a death receptor, Fas-associated death domain protein (FADD) is recruited and the death-inducing signaling complex (DISC) (for FasL/FasR, TRAIL/TRAIL-R1 or TRAIL-R2) or complex II (in the case of TNF/TNF-R1) is formed

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Conventional AD management relies on emollients or creams to maintain skin hydration or deliver therapeutics, yet these formulations often provide only transient epidermal barrier protection and short drug residence times ( in vitro safety and antimicrobial efficacy of the formulation, as well as its in vivo therapeutic potential in an AD mouse model, demonstrating enhancing skin barrier proteins, ROS scavenging, and suppression of Th2-associated inflammatory cytokines ( Figure 1 )