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The primary molecular mechanism of TB-500 involves regulation of actin dynamics through its high-affinity binding to monomeric G-actin

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Such a particular activity in angiogenesis and healing is concordant with the previous evidence that BPC 157 may directly protect endothelium (13), influence NO-system, counteract the effect of NOS-inhibitor and NO-precursor (14, 15), as well as over expression of endothelin (16)