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We also explored several other known regulators of PGC1 activity that are altered in response to 5FU treatment in HCT116, including NRF2, reactive oxygen species and PPAR, but none of these prevented PGC1-driven increases in mitochondrial mass

What the Research Has Examined Most of the interest in AHK-Cu comes from early-stage laboratory research, so it's worth being precise about what that research actually looked at - and what it didn't

doi: 10.1186/s13018-021-02771-1

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