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Sleep-Wake Regulation Early experiments associated DSIP with changes in EEG delta power, sleep spindles and the timing or stability of sleep
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Luteolin treatment inhibited cellular autophagy in the PFC, while increased in autophagic activity in the hippocampus of CUMS rats
This is different from steroid hormones, which are lipid-based and can enter cells to influence gene expression more directly
A previous cellular study has found that treatment of chondrocytes isolated from femoral condyles of patients with OA with TGF-1, a cartilage protective factor and an important regulator of cartilage homeostasis, increased the expression of HK2 and glucose transporter 1(one of the key regulators of glycolysis), thereby stimulating enhanced aerobic glycolysis and increasing the production of abnormal metabolites (78)
