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rFGF21 treatment failed to reduce mitochondrial damage and neuronal death upon SLC25A39 knockdown in primary neuron To elucidate the involvement of SLC25A39 in the protective effects of FGF21 on mitochondrial integrity and neuronal viability, we employed adenovirus-mediated siRNA technology to knock down SLC25A39 in primary neurons

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Additionally, the reliability of these findings is compromised by the small number of participants and the short duration of the treatments, which lasted only 4 and 8 weeks

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