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Beyond this direct chemical defense, NO also functions as a critical signaling molecule, modulating protein function through regulatory S-nitrosylation to actively suppress the ferroptotic cascade

Studies have also shown that -HB increases oxidative phosphorylation via a Complex II-dependent mechanism

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MMF can modulate neutrophil adhesion, recruitment, and migration through blockage of hydroxycarboxylic acid receptor 2, a receptor important for neutrophil extravasation