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Overall, these findings suggest that the rise of cellular cholesterol content favors the transition of microglia to a protective phenotype after PAMP exposure, stimulating phagocytosis and the release of neurotrophic factors via an inflammasome-dependent mechanism

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Thus, an improved understanding of the regulation of UA and its neuroprotective action in the human brain will shed further light on neurodegenerative processes and potentially identify novel targets for pharmacotherapeutic intervention

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