UbiBrowser 2.0 database indicated that NEDD4, VHL, SMURF1, and SIAH2
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[57] demonstrated that mice administered MPTP and fed KD were characterized by less motor dysfunction, dopaminergic neuron loss, and neuroinflammation than mice that only received MPTP
Disrupting glycolysis or silencing LDHA reduces H3K18la and suppresses tumor growth and metastasis in vitro and in vivo, supporting the glycolysisH3K18laTTK/BUB1B axis as a therapeutic target [90]
Furthermore, there were reductions in controlled attenuation parameter (CAP), alanine aminotransferase (ALT), and intima-media thickness (IMT) values, indicating improved liver function and reduced fat accumulation
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